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It is not for human consumption or for use in diagnostic, therapeutic, or clinical procedures

It appears that the disequilibrium of cofactors in the gdh3/gdh3 mutant leads to characteristic proline degradation in the central carbon metabolism

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We found that the mitochondria-specific CoQ potently inhibited GPX4 inhibitionmediated ferroptosis, suggesting that mitochondrial lipid redox regulates ferroptosis in AML cells