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found through experiments that compared to the TMAO group, synaptic structural damage significantly improved in the AOB group, characterized by regular synaptic morphology, enhanced vesicle distribution within the presynaptic region, and more obvious synaptic clefts

Furthermore, the central role of mTORC1 in BCAA-mediated insulin signaling suppression is validated by the finding that the mTORC1 inhibitor rapamycin can effectively reverse BCAAs-induced IR (90)

[DOI] [PubMed] [Google Scholar] 96.Longo N, Scaglia F, Wang Y

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