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This interplay is partially mediated by neuropeptides such as calcitonin gene receptor peptide (CGRP), pituitary adenylate cyclase activating polypeptide (PACAP) and/or cortical spreading depression (CSD) and involves oxidative stress, mitochondrial dysfunction, nucleotide-binding domain-like receptor family pyrin domain containing-3 (NLRP3) inflammasome formation, activated microglia, and reactive astrocytes

Consistent with these findings, our present and previous studies confirmed that endothelium-dependent vasodilation is significantly impaired in IGF- 1R KD mice [22, 44]

The intricate connections among various cell death pathways are confusing: is lipid peroxidation a direct cause of cell death, or does it initially induce one type of cell death and subsequently another [276, 277]

Hydrogen sulfide (H2S) metabolism in mitochondria and its regulatory role in energy production

On the whole, although there is no direct evidence in COVID-19 patients, PAC is likely to promote GSH depletion, especially in those population groups at higher risk (Mast et al., 2018)