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Indeed, the DNMT1/UHRF1 complex recognizes 5 mC at hemi-methylated CpGs during S phase and methylates the unmodified cytosine on the newly replicated DNA strand but does not recognize hemi-modified CpGs with oxi-mCs resulting from TET activity, thus preventing the reestablishment of symmetrical CpG methylation on the newly synthetized strand (Otani et al., 2013)

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