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*Correspondence: Mohanned Naif Alhussien, [email protected] These authors share first authorship Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

doi: 10.1111/1750-3841.14011 112

To test the hypothesis, we generated a CHAC1 knock-in (KI) mouse line with a point mutation introduced into the Chac1 genomic locus that inactivates its glutathione degradation activity

It is noteworthy that the CNS-residing NSPCs show many biochemical and morphological differences throughout life, but the origin of adult NSPCs -type B cells- has been traced from early neurodevelopmental stages (Merkle et al., 2004)

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