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Mitochondrial GSH and GSSG abundance (top and middle panels) and total intracellular GSH levels in parental HEK-293T cells and SLC25A39 knockout cells expressing a vector control or SLC25A39 cDNA

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Additionally, this proves that VCN managed to reduce the production of ECM by inhibiting the formation of myofibroblasts and epithelial-mesenchymal transitioning, probably via interfering with the TGF-1/Smad3 signaling pathway

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