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These mutations disrupt iron homeostasis by phosphorylating Rab8a, leading to the sequestration of Tf within lysosomes and potentially promoting iron accumulation and ferroptosis

Key residues 38 YGSL 41 , D 68 , R 72 , E 115 and Y 143 are responsible for high affinity substrate binding via salt bridge interaction with the glutamate (E 115 ), hydrogen bonding as well as a cation -interaction involving Y 143

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In recent years, the conversation around antioxidants has surged, largely due to their role in maintaining optimal health in an increasingly stressful world

Molecular determinants of acrylamide neurotoxicity through covalent Docking