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The primary mechanism of Semax involves significant upregulation of BDNF and nerve growth factor, which promote neuroplasticity and neuronal survival
Albert-Weissenberger, C
Despite the above mechanisms it is also possible that, to some extent, the association between AD and homocysteine simply reflects co-existing vascular disease rather than an association with pure AD pathology
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This condition is also referred to as inguinal tunnel syndrome or BernhardtRoth syndrome
