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Proviral insertion in murine lymphomas 2 (PIM2) is reported to act as an oncogene in gastric cancer, controlling apoptosis via ROS-triggered ER stress, and promoting the development of gastric cancer (72)

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Unfortunately, its effectiveness can be limited particularly during tumor recurrence by its toxicity to renal proximal tubule cells (reviewed in Zhang and Hanigan, 2003)

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In contrast, a metabolic shift toward OXPHOS is often observed during lineage commitment and differentiation, as mitochondrial activity increases to meet the energy and biosynthetic demands of specialized cell types [189, 190]