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R., Ernst H., Greaves P., Kolenda-Roberts H., Malarkey D

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Additional support for the model of GST regulation is provided by the observations that either immunodepletion of GST , or its inhibition by a rationally designed GSH-based-peptidomimetic inhibitor, - -glutamyl- S -(benzyl)cysteinyl- R ()-phenyl glycine diethyl ester (TLK199), also results in the activation of JNK

It modulates the electron transferring from the bioenergetic-derived reducing equivalents, i.e., nicotinamide adenine dinucleotide (NADH) and flavin adenine dinucleotide (FADH 2 ), through the complex I, II, and III in the electron transport chain (ETC)

doi: 10.1002/biof.5520180211