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The ELA-32 peptide, a mature form of elabela, alleviates neuronal ferroptosis, reduces cerebral infarction volume, and improves neurobehavioral deficits and cognitive dysfunction

While current conventional treatments for DKD can slow down disease progression to some extent, they come with adverse effects, increased risk of cardiovascular disease, and eventual loss of renal function

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To view a copy of this license, visit About this article Cite this article Xing, N., Du, Q., Guo, S

This local iron overload in DSCs triggers ferroptosis through reduced GSH and glutathione peroxidase four levels with the concomitant increase in lipid and cellular ROS
