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Abstract The roles of both neuroinflammation and oxidative stress in the pathophysiology of epilepsy have begun to receive considerable attention in recent years

Studies have shown that NCOA4 mediates the catabolism of ferritin according to the characteristics of the cellular environment, which regulates the mechanism of ferroptosis more significantly than directly inhibiting GPX4 to induce ferroptosis [97]

Contribution of oligomerization to PIF4s role in thermomorphogenesis Although certain TFs can bind DNA as monomers, most engage their targets as dimers or higher-order multimers 47

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A qualified healthcare provider should assess biomarker status, medication interactions, baseline detoxification capacity, and individual aging goals before recommending glutathione optimization as part of a longevity strategy