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Panels that pair flux readouts (e.g., DHE2-OH-E for O 2 , compartment-targeted HyPer/roGFP-Orp for H 2 O 2 , peroxiredoxin redox state) with damage footprints (F-isoprostanes, carbonyls/nitrotyrosine, 8-oxoG) and iron/ferroptosis metrics (labile iron pool, oxidized phospholipids, GPX4 context) provide the attribution needed to separate signalling from injury, compare studies and titrate therapy

SYRCLEs risk of bias tool for animal studies

What are the causes of B12 deficiency

Mitochondria play a central role in APAP-induced cell death and injury and therefore greater knowledge about the relationship between mitochondria with other organelles such as lysosomes can result in improved future therapeutic options for APAP-induced liver injury

Arch Biochem Biophys 754:109952