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References 1.Migicovsky Z., Sawler J., Gardner K.M., Aradhya M.K., Prins B.H., Schwaninger H.R., Bustamante C.D., Buckler E.S., Zhong G.Y., Brown P.J., et al

Nonetheless, a balanced perspective is vital

Numerous signaling pathways, such as cyclooxygenase 2 (COX-2), mammalian target of rapamycin (mTOR), mitogen-activated protein kinase (MAPK) signaling, phosphoinositide 3 kinase (PI3K), and the GATA4/p62-mediated autophagy pathways result in the stimulation of NF-B and/or C/EBP pathways, which in turn regulate the SASP secretome (81, 82)

The transcription factor Nrf2, possibly in complexes with other Jun or Maf proteins, was found to be responsible for trans -activating the human GCLC promoter via binding to ARE4 in response to numerous treatments including -NF, pyrrolidine dithiocarbamate, TBH [104,105], and insulin [106]