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Furthermore, controlling colonic ferroptosis is achievable through the Nrf2/HO- 1 signaling pathway, which suppresses the NF-B pathway and reduces the release of pro-inflammatory mediators [69]

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10.1111/j.1365-313X.2006.02746.x 16 AsgherM.KhanM

Among these, teplizumab, an anti-CD3 monoclonal antibody, has emerged as a landmark intervention capable of delaying the onset of clinical T1D in high-risk individuals (129, 130)

T2D had been associated with increased intestinal permeability, able to cause penetration of bacteria across gut barrier, resulting in metabolic endotoxaemia and determining low-grade systemic inflammation [53]