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Khorram et al
This hypothesis posits that with steatosis, the liver undergoes ER stress, oxidative stress and mitochondrial dysfunction, leading to an increase in oxidative metabolites, hepatocellular damage and secretion of a large number of inflammatory factors, which ultimately leads to further deterioration into liver disease, liver fibrosis, cirrhosis, inflammatory necrosis and other reactions (19,20)
10.3389/fphys.2020.00713 48 RaoY.ChenJ.GuoY.JiT.XieP
stability, bioavailability and, most importantly, brain endothelial transporters can also play a role
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